What's New in Neurofeedback

A Monthly Summary of News and Events

Vol. 1 No. 12 - December 1998

This newsletter is sponsored by EEG Spectrum International, Inc.,
a leader in providing clinical service and training professionals.

Past issues are available at www.eegspectrum.com/newsletter/
Information on how to subscribe or cancel a subscription appear at the end.
The opinions related in this newsletter reflect those of the author only.
Copyright (C) 1998 by EEG Spectrum International, Inc. All rights reserved.



  • Announcements  - School Program, Dissertation, Specialty Courses; New clinicians / new offices
  • In the Spotlight   - THX-1138: Coming to a School Near You
  • News & Reviews - Books, journal papers, of interest
  • Online Dialogue - Newsgroups, online news
  • Offline Dialogue - Conferences, training courses
  • Last Word               - A Commentary on the Current QEEG Controversy

  •  

    Announcements

    New Neurofeedback Clinicians / New Offices

    Charles Passler, D.C.
    80 East 11th Street, Suite 501
    New York, NY 10003
    (212) 995-5525
    Email: cpassler@mindspring.com
    
    John McManus, Ph.D.
    Abilities Center
    15100 S.W. Boones Ferry Rd, Suite 700
    Lake Oswego, OR  97035
    (503)636-0111  Fax: 977-9583
    Email: abilitiescenter@inetarena.com
    
    Elizabeth Moncrieff, M.A.
    180 Washington St.
    Norwich, CT 06360
    (860) 589-7258
    
    Fernando Augusto, Ph.D.
    Angell Street Wellness Collaborative     
    469 Angell St.                          
    Providence, RI 02906                     
    (508) 677-0493
    Mailing: P.O. Box 251
    Somerset, MA 02726
    
    Brian Carrico
    21707 Hawthorne Blvd, #103
    Torrence, CA 90503
    (310) 792-6236
    
    Ladell Lybarger, R.N.
    1221 Birch Lane
    Des Moines, IA 50315-3019
    (515) 244-1883
    
    (change of address only)
    Barry Belt, M.A., Licensed Psychologist
    A Center for Educational & Personal Development
    Floral Vale Professional Park
    503 Floral Vale Blvd
    Yardley, PA 19067
    (215) 497-0240; F-0259
    Email: BABelt@aol.com
    
    (not affiliated with EEG Spectrum International)
    Linda C. Vlay M.S.,R.N., C.S., N.P.P. A.N.P. 
    Snug Harbor Counseling Associates
    1212 Route 25A
    Stony Brook, New York  11790
    (516) 751-7474   Fax: (516) 689-7931
    Email: colleena@erols.com
    
    Attention Disorders Neurofeedback Centre (ADNC)
    Kimberly Druckman B.A., M.Ed
    201-2245 W. Broadway,
    Vancouver, B.C.,
    V6K 2E4  
    CANADA
    604-730-9600
    Email: mikmac@istar.ca
    

     


    In the Spotlight

    THX-1138: Coming to a School Near You

    by David Kaiser

    Which of the following classic science fiction films best reflects our future?

    1. 2001: A Space Odyssey
    2. Blade Runner, or
    3. THX-1138

    If you chose the third title, Lucas' first feature film from 1971, you are correct... or so the NIH Consensus Conference would have you believe. THX-1138 takes place in the 25th century. By this time the State has taken control of our mental health and everyone is drugged into a continual stupor. Loudspeakers in the home and at work constantly remind individuals to take the appropriate amount of sedatives. Avoiding one's "medication" is a crime. Near the end of the film our fate is tied to one of the men who has fought the system and failed. He's about to be arrested and "re-educated." With nowhere else to go or hide, he sits alone in a lobby, calmly awaiting the authorities. We see a line of children ride up an escalator near him. The children are noticeably subdued, passive, almost without curiousity. The camera focuses on an IV-tube strapped to each of their right arms. One of the children appears frightened and confused and approaches our man for help. His IV tube has come loose. Our man, drained of all his fight, reattaches the IV tube and with a sweet smile sends the child on his way... Perhaps this way is best for all of us, his weak smile conveys...

    Last month the National Institutes of Health held an invitation-only conference called the "Consensus Development Conference" from which they "determined" what is known and not known about the diagnosis and treatment of Attention Deficit Hyperactivity Disorder. The Conference was sponsored by the National Institute on Drug Abuse, the National Institute of Mental Health, and the NIH Office of Medical Applications of Research (and assorted co-sponsors). This conference brought together national and international experts in the fields of relevant medical research and health care as well as representatives from the public. EEG Spectrum International was contacted by one of the participants to provide him information about alternative treatments -- although none of this information appears to have trickled into the consensus. A draft of the consensus statement can be found at http://odp.od.nih.gov/consensus/cons/110/110_intro.htm. But here are some of the key points of "agreement":

    First, attention deficit hyperactivity disorder (ADHD) is recognized as the most commonly diagnosed behavioral disorder of childhood, affecting 3 to 5 percent of school-age children. This translates to about 3,000,000 children. The core symptoms of ADHD include a "developmentally inappropriate level of attention and concentration and developmentally inappropriate levels of activity, distractibility, and impulsivity. Children with ADHD usually have pronounced difficulties and impairment resulting from the disorder across multiple settings in home, at school, and with peers as well as resultant long-term adverse effects on later academic, vocational, social-emotional, and psychiatric outcomes. "

    Some people claim ADHD doesn't exist at all, others that it is overdiagnosed or a catch-basin for other childhood problems. The rate of increase in the number of diagnosed ADHD children would suggest that we are in an ADHD epidemic, one that -- if the rate does not subside -- will include the entire school-age population in the US within 10 to 15 years. Treatment of this condition is most controversial as respectable scientists call for pharmacological solutions to childhood.

    One of the major controversies regarding ADHD concerns the use of psychostimulants to treat the condition. Psychostimulants, including amphetamine, methylphenidate, and pemoline, are by far the most widely researched and commonly prescribed treatments for ADHD. The use of methylphenidate and amphetamine nationwide has increased six-fold since 1990, intensifying the concerns about use, overuse, and abuse of this stimulant. According to the federal Drug Enforcement Administration, more than one in every 30 Americans between 5 and 19 years old has a prescription for Ritalin. Of particular concern to parents is that the long-term effects of taking Ritalin are not known. The manufacturers of Ritalin warn: SUFFICIENT DATA ON THE SAFETY AND EFFICACY (EFFECTIVENESS) OF LONG TERM USE OF RITALIN IN CHILDREN ARE NOT YET AVAILABLE." (Quoted from CIBA Pharmaceutical Company in a product information release.)

    The consensus statement addressed the following key questions:

    1. What is the scientific evidence to support ADHD as a disorder?
    2. What is the impact of ADHD on individuals, families, and society?
    3. What are the effective treatments for ADHD?
    4. What are the risks of the use of stimulant medication and other treatments?
    5. What are the existing diagnostic and treatment practices, and what are the barriers to appropriate identification, evaluation, and intervention?
    6. What are the directions for future research?

    In this article I will only address points 1,3, and 4.

    1. What Is the Scientific Evidence To Support ADHD as a Disorder?

    "The diagnosis of ADHD can be made reliably using well-tested diagnostic interview methods. However, we do not have an independent, valid test for ADHD, and there are no data to indicate that ADHD is due to a brain malfunction. "

    This is the kind of wording which makes me dislike scientists (excluding myself and my friends, that is). It is arrogance dressed up as fact. There are no data..." Unless the statement is purposely unclear or turns on a limited interpretation of malfunction it would appear to say that there is no data showing that ADHD children differ from non-ADHD on any measure of brain function. I know of a dozen papers off the top of my head which refute this point. Doing a search of just the past two years I found the following papers:

    Clarke AR, Barry RJ, McCarthy R, Selikowitz M
    EEG analysis in Attention-Deficit/Hyperactivity Disorder: a comparative study of two subtypes.
    Psychiatry Res 1998 Oct 19;81(1):19-29

    Boutros N, Fristad M, Abdollohian A
    The fourteen and six positive spikes and attention-deficit hyperactivity disorder.
    Biol Psychiatry 1998 Aug 15;44(4):298-301

    Lazzaro I, Gordon E, Whitmont S, Plahn M, Li W, Clarke S, Dosen A, Meares R
    Quantified EEG activity in adolescent attention deficit hyperactivity disorder.
    Clin Electroencephalogr 1998 Jan;29(1):37-42

    Amen DG, Carmichael BD
    High-resolution brain SPECT imaging in ADHD.
    Ann Clin Psychiatry 1997 Jun;9(2):81-6

    Strandburg RJ, Marsh JT, Brown WS, Asarnow RF, Higa J, Harper R, Guthrie D
    Continuous-processing--related event-related potentials in children with ADHD.
    Biol Psychiatry 1996 Nov 15;40(10):964-80

    Chabot RJ, Serfontein G
    Quantitative electroencephalographic profiles of children with ADD.
    Biol Psychiatry 1996 Nov 15;40(10):951-63

    Kuperman S, Johnson B, Arndt S, Lindgren S, Wolraich M
    Quantitative EEG differences in a nonclinical sample of children with ADHD and undifferentiated ADD.
    J Am Acad Child Adolesc Psychiatry 1996 Aug;35(8):1009-17

    Vaidya CJ, Austin G, Kirkorian G, Ridlehuber HW, Desmond JE, Glover GH, Gabrieli JDE
    Selective effects of methylphenidate in ADHD: An fMRI study.
    Proc Natl Acad Sci U S A 1998 Nov 24;95(24):14494-9
    Research from the world's best scientific laboratories and universities -- Stanford University, UCLA, Yale, NYU -- and most find the same results -- frontal slowing or decreased activity depending upon the measure (QEEG, PET, SPECT).

    How can the Conference dismiss 80 or so papers (since 1996) which find differences between ADHD and non-ADHD brains? Was I reading a scientific report or a nifty propaganda piece? How can they draw firm conclusions about certain points with little evidence (see below) and ignore results wholesale from others? Swanson et al (1998; Current Opinions in Neurobiology, 8(2):263-71) reported that certain regions of the frontal lobes (anterior superior and inferior) and basal ganglia (caudate nucleus and globus pallidus) are about 10% smaller in ADHD groups than in controls. Swanson was a speaker at the conference but still there was "no data" of neurobiological impairment in ADHD. Zametkin and Liotta (1998) of NIMH performed a comprehensive review of the neurobiological basis of attention-deficit/hyperactivity disorder and came to the conclusion that CNS abnormalities are associated with ADHD. How can respected scientists be overlooked and their work dismissed entirely?

    3. What Are the Effective Treatments for ADHD?

    "A wide variety of treatments have been used for ADHD including, but not limited to, various psychotropic medications, psychosocial treatment, dietary management, herbal and homeopathic treatments, biofeedback, meditation, and perceptual stimulation/training... Of these treatment strategies, medications and psychosocial interventions have been the major focus of research and the only ADHD research I am aware of funded by NIH or NIMH sources."

    They remind us that stimulant treatments do not "normalize" the entire range of behavior problems, and children under treatment still manifest a higher level of some behavior problems than normal children. And, notably, "there is little improvement in academic achievement or social skills" with stimulant treatment. Also, there are no long-term studies testing stimulants or psychosocial treatments nor information on long-term outcomes of medication-treated ADHD individuals in terms of educational and occupational achievements, or other areas of social functioning. Biofeedback is mentioned in passing and at no place do they say that it deems more research. However,one of the most respective and prolific ADHD researchers was prepared to spend five years at the peak of his career to study neurofeedback's effect on ADHD children. Neurofeedback alone! I doubt a respected scientist would have planned such an effort had medications and psychosocial treatments been sufficient or even desirable for the majority of ADHD children.

    4. What Are the Risks of the Use of Stimulant Medication and Other Treatments?

    "Although little information exists concerning the long-term effects of psychostimulants, there is no conclusive evidence that careful therapeutic use is harmful. When adverse drug reactions do occur, they are usually related to dose."

    Perhaps this is true (though "no conclusive evidence" is lawyerspeak for my client is guilty but you can't prove it). I think it is true to the extent that people are placing too much burden on stimulant medication to solve their problems. Trying to pass off various conduct and emotional problems as attentional issues underlies the overdiagnosing of ADHD. Dr. Breggin, who also attended the conference, mentions that a multimillion dollar program is now promoting the use of Ritalin for the control of disruptive behavior. THX-1138 is coming...

    The following effects of stimulant treatment are thus judged as non-harmful: "decreased appetite and insomnia, negative effects on growth rate, and motor and vocal tics: The growth rate change concerns me the most because they qualify this obviously unusual side effect with the following "but ultimate height appears not to be affected." How do they know this? How arrogant to make a statement like this with little or no data! Rao et al (1998) found a small but significant effect on growth due to stimulant medication in their patient population under study. Spencer et al (1998) reported the small but significant differences in height between children with and without ADHD but concluded this effect appears to be mediated by ADHD and not by its treatment. Six years earlier Spencer concluded that children "treated (chronically) with MPH sustained height deficits that attained statistical significance". So we have conflicting results. Maybe there is no permanent effect -- but the jury is still out and the statement should be qualified accordingly. They say "Further, it is concluded that there is a low probability of long term effects on human body stature when the minimal therapeutic dose is used in clinical practice. " This is the most telling aside as it appears that fewer and fewer children are being prescribed the "minimal dose".

    Another issue is abuse and addiction. When children pop psychotropic pills every 90 minutes or so, do you think they are more or less likely to try other mind-altering substances? NIH says no effect -- no more or no less. "There is little evidence that current levels of production have had a substantial effect on abuse. " Then why is methylphenidate one of the most frequently reported controlled pharmaceuticals stolen from licensed handlers. (Source: Dept of Justice) . And what should we make of the increasing reports of Ritalin-abuse on college campuses and secondary schools (see http://www.student.com/article/ritalin , The Ritalin Racket)? In 1994, a national survey indicated that more seniors in the U.S. abused Ritalin than are prescribed it legitimately. Hmmm.... According to the Physician's Desk Reference Guide, the standard text for medical reference, "chronic abuse [of Ritalin] can lead to marked tolerance levels and psychological dependence with varying degrees of abnormal behavior." Hmmm, again.... In the consensus statement, we are cautioned that "there is a need to be vigilant in monitoring the national indices of use and abuse among high school seniors and Drug Abuse Warning Network (DAWN) emergency room reports." According to DAWN statistics, an estimated 2700 emergency room mentions of methylphenidate occurred between 1990 and 1993. And finally, in March of 1995, two deaths in Mississippi and Virginia were associated with students giving and selling their medication to classmates who were crushing and snorting the powder like cocaine (Source: Drug Enforcement Administration press release http://www.usdoj.gov/dea/pubs/pressrel/pr951020.htm. Hmmm, very strange for a non-addictive substance.

    Much of the report is clear and reasonable, based on scientific evidence, but here and there a bit of dogma seemed to be dressed up in academic garb.

     


     

    News & Reviews

    NEW BOOKS

     

    The Hyperactivity Hoax: How to Stop Drugging Your Child and Find Real Medical Help
      by Sydney Walker
      St Martin's Press, 288 pp, $17

    Sydney Walker III, M.D., a board-certified neurologist and psychiatrist and Director of the Southern Cal Neuropsychiatric Institute, has treated "ADHD" children for more than 30 years. He argues that a wide range of problems -- metabolic and genetic disorders, heart conditions, infections, anemia, hearing and vision problems, and toxic exposure can produce supposed ADHD symptomatology (e.g., hyperactive behavior, inattention) and masquerade as the ADHD. Accordingly, he cautions against the "label-and-drug" fad that has currently swept through U.S. child psychiatry the last decade or so. Children are too often placed on "powerful and potentially harmful mind-altering drugs without the benefit of any real medical diagnosis." He offers step-by-step advice about determining what form of care your child needs.

    For more information, see http://www.amazon.com/exec/obidos/ASIN/0312192878/

     

    Last Resort: Psychosurgery and the Limits of Medicine
      by Jack D. Pressman

    In 1935 Egas Moniz, a Portuguese neurosurgeon and future Nobel laureate, partially destroyed small areas in the frontal lobes on 13 depressed patients and 7 schizophrenic patients, resulting in marked improvement in many of the depressed patients. Removing malfunctioning brain was out of the gate... Some 20,000 psychiatric patients later, today we no longer look so highly on frontal lobotomies. They are no longer done and the entire episode has become an embarrassment to psychiatrists and neurologists who would prefer to dismiss it as a one-time aberration. (Will drugging our active children into sitting still be viewed better in 40 years?)

    For more information, see http://www.amazon.com/exec/obidos/ASIN/0521353718/

    Drug Addiction and Its Treatment: Nexus of Neuroscience and Behavior
      by Bankole A. Johnson & John D. Roache (eds) The editors examine the behavioral and biological processes involved in drug addiction and suggests ways of integrating behavioral and pharmacological treatments. The book is divided into four sections: behavior, neurobiology, trends in neuroscience, and treatment applications. The third section in particular may be of interest to neurofeedback clinicians as they describe state-of-the-art research techniques for studying the neurobiology of drug addiction including topographical brain mapping.

    For more information, see http://www.amazon.com/exec/obidos/ASIN/0397517645/

     

    Discoveries in the Human Brain: Neuroscience Prehistory, Brain Structure, & Function
      by Louise H. Marshall & Horace W. Magoun
      Humana Press, 336 pp., $60

    The history of the brain research and the emergence of modern neuroscience are chronicled from the first findings of gross neuroanatomy in the ancient world to present-day neural networks and brain modeling. Along the way the people and events which gradually produced today's understanding of brain anatomy and physiology are presented, often with quotations from primary sources.

    For more information, see http://www.amazon.com/exec/obidos/ASIN/0896034356/

     


    JOURNAL PAPERS

    Neurofeedback treatment of pseudoseizure disorder.
      Swingle PG
      [ Dept of Psychiatry, Harvard Medical School, USA. ]
      Biol Psychiatry 1998 Dec 1;44(11):1196-9

    It took only 30 years for someone at Harvard to take note of Sterman's remarkable epilepsy research. Pretty quick of them ... Once per week three patients underwent SMR training, along with psychotherapy -- always an effective way to keep neurons from kindling. Swingle reported a correlation between reduced seizure activity and smaller theta-SMR ratios and he concluded that neurofeedback -- in conjunction with psychotherapy! -- is an "effective adjunctive treatment for pseudoseizure disorder".

    The functional neuroanatomy of major depression: an fMRI study using an emotional activation paradigm.
      Beauregard M, Leroux JM, Bergman S, Arzoumanian Y, Beaudoin G, Bourgouin P, Stip E
      [ Dept de Radiologie, Faculte de Medecine, Universite de Montreal, Quebec, Canada. ]
      Neuroreport 1998 Oct 5;9(14):3253-8

    Viewing material likely to induce sadness activated medial and inferior prefrontal cortex, temporal cortex, and the caudate in both depressed and normal subjects. However, in depressed subjects, emotional material produced a greater activation in left medial prefrontal cortex and in the right cingulate gyrus compared to controls, which suggests that these two cortical regions might be part of a network implicated in the pathophysiology of major depression.

    EEG analysis in ADHD: a comparative study of two subtypes.
      Clarke AR, Barry RJ, McCarthy R, Selikowitz M
      [ Dept of Psychology, University of Wollongong, NSW, Australia ]
      Psychiatry Res 1998 Oct 19;81(1):19-29

    ADHD children exhibit greater theta activity and reduced alpha and beta activity compared to controls. Inattentive ADHD subtype differed from the Combined subtype in the same measures, appearing to be closer to controls. The authors argue that these results support a maturational lag model of the central nervous system in Attention Deficit/Hyperactivity Disorder and that subtype differences reflect severity rather than dysfunctional differences.

    Attention deficit diagnosis and care uncertain
      Various
      NIH Consensus Conference

    According to the National Institutes of Health (NIH), we still don't know the best way to treat or even diagnose attention deficit disorder in children. More than a million US children now take powerful drugs to remediate a diagnosis they may or may not have. The recent consensus statement shows that there is not as much consensus among ADHD researchers as one would think.

    Five Top Warning Signals Of Depression Identified
      Cheryl Carmin & John Klocek
      [ University of Illinois at Chicago, ]
      The International Journal of Psychiatry in Medicine, Fall 1998

    Approximately 90% of depressed individuals report the following five symptoms: Less enjoyment from usual activities; Disappointment with self; Hopelessness; Irritability ; Difficulty sleeping
    Depressed women most often report self-disappointment and irritability whereas depressed men most often reported dissatisfaction and/or difficulty sleeping. A few simple questions or attention to these complaints during an office visit might suggest an underlying mood disorder (confirmed with further screening).

    Brain's Ability To "Rewire" Itself After Strokes Or Injuries
    (Large-scale sprouting of cortical connections after peripheral injury in adult macaque monkeys.)

      Florence SL, Taub HB, Kaas JH
      [ Dept of Psychology, Vanderbilt University, 301 Wilson Hall, Nashville, TN 37240 sherre.l.florence@vanderbilt.edu ]
      Science 1998 Nov 6;282(5391):1117-21

    The adult brain has a surprisingly robust built-in capacity for change, creating the possibility for innovative treatments for brain disorders. A Vanderbilt study shows that neurons in the adult brain can sprout new axons which can travel and make contact with new targets at distant sites in the brain. New cell growth was believed to be initiated in response to massive change in activity patterns, not from the injury itself. This suggests that natural processes (such as neurofeedback) can coax flexibility out of the adult brain -- in contrast to research where chemicals are administered to facilitate cell growth.

     

    Online Dialogue
    Online News

    Clinics Online

    More clinicians have added information online. Sixty-two offices currently online.

     


     

    Offline Dialogue

    EEG Biofeedback Training Course for Professionals

    EEG Spectrum International presents the emerging field from a perspective well-grounded in clinical phenomenology, but it is also based on a neurophysiological model of efficacy originally proposed by M. Barry Sterman, Ph.D., and recently augmented by Andrew Abarbanel, M.D. The clinical database and the model combine to yield a variety of protocols which have proven to be effective in the treatment of behavior, mood, pain, attention and learning problems, seizure disorder and the consequences of brain injury. A coherent picture emerges about how protocol choices may be made in the face of complex presenting symptoms.

    The training also includes the all-important practicum section which focuses on actual operation and use of instrumentation. Additionally, opportunity is provided outside the formal curriculum for practitioners to experience training on themselves as well as discussing professional issues, marketing, and the particulars of establishing a financially successful practice based on this modality.

    Faculty: Siegfried Othmer, Ph.D., BCIAC; Susan Othmer, BCIAC; M. Barry Sterman, Ph.D.; Nancy White, Ph.D. ; Pat Fields, Psy.D.; Martin Wuttke, BCIAC; William Scott, BSW, CCDP


    LOCATION DATES
    Encino, CA     (Adv. Practicum) Dec 12-13, 1998
    Encino, CA     (Specialty Course) Jan 7-11, 1999
    San Diego, CA     Jan 21-25, 1999
    Orlando, FL     Feb 18-22, 1999

    TOPICS COVERED
    EEG Biofeedback Theory: Neurophysiological Basis; Research History
    Clinical Applications: Assessment, Protocol Selection, Practicum & Case Review
    Specialty Applications: For Behavior Modification & Performance Enhancement
    COST (5-Day Course): $995.00 (** excluding Specialty courses)
    Additional Attendees from Same Facility: 30% discount     Reattendees: $200.00

    To enroll, contact Dennis Campbell or call EEG Spectrum International at 800-789-3456 or (818) 788-2083. Or email denniscampbell@eegspectrum.com Please include your name and phone number in all email messages.



    Conferences for Neurofeedback Clinicians & Researchers

    CONFERENCELOCATION DATES
    FutureHealth 1999 (see below) Palm Springs, CA February 5-9, 1999
    AAPB 1999 Vancouver, BC April 7-11, 1999


    1999 FutureHealth Conference: Palm Springs, February 5-9, 1999

    For additional information and updates: bio@Futurehealth.org

    In a related note, the FutureHealth 1998 abstracts are online at http://www.futurehealth.org/97eegab1.htm

     


     

    Last Word

    A Commentary on the Current QEEG Controversy

    In a posting to the PsyPhy list server, Ted LaVaque asserted some time ago that on the basis of the QEEG there may be no good reason to do beta/SMR training at all. Here is Siegfried Othmer's reply ( SigOthmer@EEGSpectrum.com):
    "The field of neurofeedback is probably between $50 and $100 million in terms of annual services delivered around the world. Probably the majority of this is in the realm of SMR/beta training-perhaps 70% or more. This work involves more than a thousand clinicians plying their trade. Do all these folks not know what they are doing, and are they duping the public and misleading themselves, and imagining the amazing results that they are claiming? Of course not, but it is revealing that a researcher is apparently still capable of entertaining such a hypothesis thirty years after Sterman demonstrated seizure reduction in cats with SMR-training in controlled studies.
    If on the other hand La Vaque's judgment is mistaken, what would that indicate? It would be another among numerous historical instances of misinterpretation of the implications of the QEEG. Just because the QEEG does not change grossly and predictably in the training band (in the SMR-beta range) does not mean that a challenge to brain function in that frequency range is not useful. Rather, it means that the functional brain constrains frequency amplitudes in these frequency ranges rather precisely -- not to a single value, but to what the brain needs it to be at a particular moment. If we assume a functional role for the instantaneous degree of rhythmicity in regulating both local activation and central arousal, then precise control of such amplitudes would follow. The brain simply won't allow an arbitrary change in EEG parameters because that would have profound functional implications! So there is no contradiction here at all.
    So if we have one individual who responds nary at all in terms of beta amplitudes, and another who responds profoundly, are we to conclude that the second was somehow the better, more compliant and more successful subject? More likely, the latter simply came with the much less stable brain!
    One of the first things we did after getting into this field was to track changes in theta, beta, and in theta-beta ratios in our subjects. After plotting up results for some 167 clients, we found that most subjects didn't change a great deal at all (80% showed changes less than +-20%) and the changes were about equally distributed between upward and downward changes! We showed these results at AAPB in 1992, and they were predictably dismissed as heresy at the time. We have not allowed our work to live or die by that parameter since (actually, we never did, and neither did Barry Sterman).
    Incidentally, this statement should not be seen as a contradiction of what Lubar was claiming at the time, either. Lubar pre-selected his subjects to have high theta-beta ratios at the outset of training. Under those circumstances, some normalization of that parameter is expected to occur, and does so as well at our hands. Where we differed with Joel is with respect to the requirement of pre-selection. Whereas such pre-selection may have uncovered a class of children who were particularly likely to respond favorably to the training (the head-injured most prominently among them), it was not capable of distinguishing those who would be responsive to the training from those who would not be.

    -Siegfried Othmer, Ph.D.


    Future LAST WORDS may contain feedback from readers of this newsletter. Send your feedback to webmaster@eegspectrum.


    Subscription Information

    Each month you will receive this newsletter via email. Each issue includes reviews of recent publications relevant to neurofeedback, updates on new and ongoing research studies, reports in the media, and schedules of upcoming training courses and conference dates. To unsubscribe from this newsletter, email webmaster@eegspectrum.com and include in the subject line of your message "unsubscribe newsletter"

    If you care to contribute to the list, email the newsletter editor at dakaiser@eegspectrum.com at any time during the month. Include in your email relevant articles, abstracts, web addresses, etc. Contributions may be edited for inclusion in the newsletter and not every contribution may appear in the monthly newsletter